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- Over 500 years family tradition /  Over 35 years experiences -

[ 10 years Acupuncture Training Under Grandfather in South Korea ]  
 [ The Highest Level of Academic Degree in Acupuncture and Oriental Medicine ] 

Dr. Kim's initial training was passed down to him by his grandfather who was the latest in a long line of family members who were Acupuncturists and MD's (Neurosurgeons/Heart Surgeons/OB-GYN's) in South Korea. Dr.Kim has a Bachelor Degree in science, Master Degree in Acupuncture & Chinese herbology, and Doctorate Degree in Oriental Medicine(Neuro-Acupuncture and Chinese Herbology). 

" When I was 11 years old, My grandfather taught me how to
 insert needles into the body.
I was so excited to learn it. My grandfather's techniques were 
unique, spirited, and delicate.  
I am still following his Journey and developing New Techniques. "

Dr.Kim always goes out of his way to help patients.
- Address the cause, not just the symptoms -
RedAcu.Com
Promoting Self-Healing
[ URGENT CARE ]

Professional Organization 
  National Certification Commission for Acupuncture and Oriental Medicine
  Texas medical board of examiners 
  Texas State Board of Acupuncture Examiners
  Texas Association of Acupuncture and Oriental Medicine 
  American College of Clinical Thermology
  TaeKwonDo 3 Degrees Black Belt 
  Tai Chi and Qi Gong Master 

Medical Achievement
  International Hippocrates Award

Military Services
  Republic of Korea Army (DMZ, 1989 - 1991)

Home School / Family Medical Training in South Korea 
  10 years under Maternal-Grandfather (Acupuncturist, Professor) in South Korea
  Additional training under Maternal-Uncles ( Neurosurgeon, Cardiothoracic surgeon)
  Additional training under Maternal-Aunts (OB/GYN, IVF specialists) 

Public / Private School Education
  Bachelor / Master Degree in Science, . . . .   
  Master Degree in Acupuncture & Herbology  
  Doctorate Degree in Oriental Medicine (Acupuncture & Herbology)           Cl

    

"Dr. Hyun Jong Kim has been selected as one of IBC's Leading Health Professionals of the World 2014, 2015, 2016, 2017, 2018, 2019.  Many thousands of biographies from a wide variety of sources are investigated by the Research and Editorial Departments of the International Biographical Center each year for inclusion on our renowned reference books and for consideration for our Awards Program. A select few are those of individuals who, in our belief, have made a significant enough contribution in their field to engender influence on a local, national or international basis. Dr. Kim is one of these contributors to excellence. Dr. Kim has, through constant efforts, maintained a standard worth rewarding. Therefore, as a noted and eminent professional in the health profession Dr. Kim has now been considered and nominated for recognition by the IBC. Ratification of this nomination by the Awards Board is now complete and it is therefore my great honor to name Dr.Kim as a member of the IBC." [ Nicholas S. Law, Director General, International Biographical Center, Cambridge, England ]
LEADING HEALTH PROFESSIONALS OF THE WORLD
Best of The World 2014, 2015, 2016, 2017, 2018, 2019. 
Dr. Hyun Jong Kim L.Ac., O.M.D., Ph.D.
" There is nothing to compare with your health. "
25222 Grogan's Mill Road, The Woodlands, Texas 77380
www.RedAcu.com


Continuing Medical Education : Provides Better Care to Patients.

Photo ( The Woodlands, Texas, 2006 ) : Dr.Kim with Patient (Korean War Veteran)  
Harvard Medical School Continuing Medical Education (CME) offers an extensive, best-in-class catalog of continuing education activities that serve the diverse educational needs of healthcare providers. It promotes quality improvement in clinical practice and health care, medical research, and lifelong learning.
American College of Acupuncture & Oriental Medicine Continuing Acupuncture Education (CAE) is open only to Acupuncturists.
TEXT OR CALL 
832) 660 4838 
The articles which Dr. Kim would like to share with patients
Solving a biological puzzle: How stress causes gray hair

HSCRB (Harvard : Department of Stem Cell and Regenerative Biology) scientists uncover link between the nervous system and stem cells that regenerate pigment
By Jessica Lau | January 22, 2020


​When Marie Antoinette was captured during the French Revolution, her hair reportedly turned white overnight. In more recent history, John McCain experienced severe injuries as a prisoner of war during the Vietnam War — and lost color in his hair.

For a long time, anecdotes have connected stressful experiences with the phenomenon of hair graying.

Now, for the first time, HSCRB scientists have discovered exactly how the process plays out: stress activates nerves that are part of the fight-or-flight response, which in turn cause permanent damage to pigment-regenerating stem cells in hair follicles.

The study, published in Nature, advances scientists’ knowledge of how stress can impact the body.

“Everyone has an anecdote to share about how stress affects their body, particularly in their skin and hair — the only tissues we can see from the outside,” said senior author Ya-Chieh Hsu, the Alvin and Esta Star Associate Professor of Stem Cell and Regenerative Biology. “We wanted to understand if this connection is true, and if so, how stress leads to changes in diverse tissues. Hair pigmentation is such an accessible and tractable system to start with — and besides, we were genuinely curious to see if stress indeed leads to hair graying. ”

Narrowing down the culprit

Because stress affects the whole body, researchers first had to narrow down which body system was responsible for connecting stress to hair color. The team first hypothesized that stress causes an immune attack on pigment-producing cells. However, when mice lacking immune cells still showed hair graying, researchers turned to the hormone cortisol. But once more, it was a dead end.

“Stress always elevates levels of the hormone cortisol in the body, so we thought that cortisol might play a role,” Hsu said. “But surprisingly, when we removed the adrenal gland from the mice so that they couldn’t produce cortisol-like hormones, their hair still turned gray under stress.”

After systematically eliminating different possibilities, researchers honed in on the sympathetic nerve system, which is responsible for the body’s fight-or-flight response.

Sympathetic nerves branch out into each hair follicle on the skin. The researchers found that stress causes these nerves to release the chemical norepinephrine, which gets taken up by nearby pigment-regenerating stem cells.

Permanent damage

In the hair follicle, certain stem cells act as a reservoir of pigment-producing cells. When hair regenerates, some of the stem cells convert into pigment-producing cells that color the hair.

Researchers found that the norepinephrine from sympathetic nerves causes the stem cells to activate excessively. The stem cells all convert into pigment-producing cells, prematurely depleting the reservoir.

“When we started to study this, I expected that stress was bad for the body — but the detrimental impact of stress that we discovered was beyond what I imagined,” Hsu said. “After just a few days, all of the pigment-regenerating stem cells were lost. Once they’re gone, you can’t regenerate pigments anymore. The damage is permanent.”

The finding underscores the negative side effects of an otherwise protective evolutionary response, the researchers said.

“Acute stress, particularly the fight-or-flight response, has been traditionally viewed to be beneficial for an animal’s survival. But in this case, acute stress causes permanent depletion of stem cells,” said postdoctoral fellow Bing Zhang, the lead author of the study.


Elaborate sympathetic innervation (magenta) around pigment-regenerating stem cells (yellow). Acute stress induces hyperactivation of the sympathetic nervous system to release large amount of the neurotransmitter norepinephrine. Norepinephrine drives rapid depletion of pigment-regenerating stem cells and hair graying. Credit: Hsu Laboratory, 
Harvard University
Infographic depicting how stem cells are depleted in response to stress, causing hair to turn gray in mice. Credit: Judy Blomquist, Harvard University
Answering a fundamental question

To connect stress with hair graying, the researchers started with a whole-body response and progressively zoomed into individual organ systems, cell-to-cell interaction and, eventually, all the way down to molecular dynamics. The process required a variety of research tools along the way, including methods to manipulate organs, nerves, and cell receptors.

“To go from the highest level to the smallest detail, we collaborated with many scientists across a wide range of disciplines, using a combination of different approaches to solve a very fundamental biological question,” Zhang said.

The collaborators included Isaac Chiu, assistant professor of immunology at Harvard Medical School who studies the interplay between nervous and immune systems.

“We know that peripheral neurons powerfully regulate organ function, blood vessels, and immunity, but less is known about how they regulate stem cells,” Chiu said.
“With this study, we now know that neurons can control stem cells and their function, and can explain how they interact at the cellular and molecular level to link stress with hair graying.”

The findings can help illuminate the broader effects of stress on various organs and tissues. This understanding will pave the way for new studies that seek to modify or block the damaging effects of stress. Harvard’s Office of Technology Development has filed a provisional patent application on the lab’s findings and is engaging prospective commercial partners who may be interested in clinical and cosmetic applications.

“By understanding precisely how stress affects stem cells that regenerate pigment, we’ve laid the groundwork for understanding how stress affects other tissues and organs in the body,” Hsu said. “Understanding how our tissues change under stress is the first critical step towards eventual treatment that can halt or revert the detrimental impact of stress. We still have a lot to learn in this area.”
Flu vaccination linked to 40% reduced risk of Alzheimer's disease
by University of Texas Health Science Center at Houston

People who received at least one influenza vaccine were 40% less likely than their non-vaccinated peers to develop Alzheimer's disease over the course of four years, according to a new study from UTHealth Houston.

Research led by first author Avram S. Bukhbinder, MD, a recent alumnus of McGovern Medical School at UTHealth Houston, and senior author Paul. E. Schulz, MD, the Rick McCord Professor in Neurology at McGovern Medical School, compared the risk of Alzheimer's disease incidence between patients with and without prior flu vaccination in a large nationwide sample of U.S. adults aged 65 and older.

An early online version of the paper detailing the findings is available in advance of its publication in the Aug. 2 issue of the Journal of Alzheimer's Disease.

"We found that flu vaccination in older adults reduces the risk of developing Alzheimer's disease for several years. The strength of this protective effect increased with the number of years that a person received an annual flu vaccine—in other words, the rate of developing Alzheimer's was lowest among those who consistently received the flu vaccine every year," said Bukhbinder, who is still part of Schulz's research team while in his first year of residency with the Division of Child Neurology at Massachusetts General Hospital. "Future research should assess whether flu vaccination is also associated with the rate of symptom progression in patients who already have Alzheimer's dementia."

The study—which comes two years after UTHealth Houston researchers found a possible link between the flu vaccine and reduced risk of Alzheimer's disease—analyzed a much larger sample than previous research, including 935,887 flu-vaccinated patients and 935,887 non-vaccinated patients.

During four-year follow-up appointments, about 5.1% of flu-vaccinated patients were found to have developed Alzheimer's disease. Meanwhile, 8.5% of non-vaccinated patients had developed Alzheimer's disease during follow-up.

These results underscore the strong protective effect of the flu vaccine against Alzheimer's disease, according to Bukhbinder and Schulz. However, the underlying mechanisms behind this process require further study.

"Since there is evidence that several vaccines may protect from Alzheimer's disease, we are thinking that it isn't a specific effect of the flu vaccine," said Schulz, who is also the Umphrey Family Professor in Neurodegenerative Diseases and director of the Neurocognitive Disorders Center at McGovern Medical School. "Instead, we believe that the immune system is complex, and some alterations, such as pneumonia, may activate it in a way that makes Alzheimer's disease worse. But other things that activate the immune system may do so in a different way—one that protects from Alzheimer's disease. Clearly, we have more to learn about how the immune system worsens or improves outcomes in this disease."

Alzheimer's disease affects more than 6 million people living in the U.S., with the number of affected individuals growing due to the nation's aging population. Past studies have found a decreased risk of dementia associated with prior exposure to various adulthood vaccinations, including those for tetanus, polio, and herpes, in addition to the flu vaccine and others.

Additionally, as more time passes since the introduction of the COVID-19 vaccine and longer follow-up data becomes available, Bukhbinder said it will be worth investigating whether a similar association exists between COVID-19 vaccination and the risk of Alzheimer's disease.



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